Max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. On the flip side, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes through the inhibition of FAK implies elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by greater pFAK localization in 5-HT https://hugop653teq4.azuria-wiki.com/user